Last five years | All
2019 2018 2017 2016 2015
| 2019 Original Articles | |||
| o | Hillebrand S, Schanda K, Nigritinou M, Tsymala I, Böhm D, Peschl P, Takai Y, Fujihara K, Nakashima I, Misu T, Reindl M, Lassmann H, Bradl M (2019) Circulating AQP4-specific auto-antibodies alone can induce neuromyelitis optica spectrum disorder in the rat Acta Neuropathol, 137(3): 467-485 | ||
| o | Wimmer I, Scharler C, Zrzavy T, Kadowaki T, Mödlagl V, Rojc K, Tröscher A, Kitic M, Ueda S, Bradl M, Lassmann H (2019) Microglia pre-activation and neurodegeneration precipitate neuroinflammation without exacerbating tissue injury in experimental autoimmune encephalomyelitis Acta Neuropathologica Communications, 7 (1): 14 | ||
| 2018 Original Articles | |||
| o | Lemke A, Ferguson J, Gross K, Penzenstadler C, Bradl M, Mayer R, Gerner C, Redl H, Wolbank S (2018) Transplantation of human amnion prevents recurring adhesions and ameliorates fibrosis in a rat model of sciatic nerve scarring Acta Biomater, 66: 335–349 | ||
| 2018 Reviews | |||
| o | Bradl M, Reindl M, Lassmann H (2018) Mechanisms for lesion localization in neuromyelitis optica spectrum disorders Curr Opin Neurol, 31(3): 325-333 | ||
| 2017 Original Articles | |||
| o | Lemke A, Penzenstadler C, Ferguson J, Lidinsky D, Hopf R, Bradl M, Redl H, Wolbank S, Hausner T (2017) A novel experimental rat model of peripheral nerve scarring: reliably mimicking post-surgical complications and recurring adhesions Dis Model Mech, 10(8): 1015-1025 | ||
| o | Peschl P, Schanda K, Zeka B, Given K, Böhm D, Ruprecht K, Saiz A, Lutterotti A, Rostásy K, Höftberger R, Berger T, Macklin W, Lassmann H, Bradl M, Bennett J, Reindl M (2017) Human antibodies against the myelin oligodendrocyte glycoprotein can cause complement-dependent demyelination J Neuroinflammation, 14 (1): 208 | ||
| 2017 Reviews | |||
| o | Zeka B., Lassmann H., Bradl M. (2017) Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder Clinical and Experimental Neuroimmunology, 8: 3-7 | ||
| o | Lassmann H, Bradl M. (2017) Multiple sclerosis: experimental models and reality Acta Neuropathol, 133(2): 223-244 | ||
| o | Peschl P, Bradl M, Höftberger R, Berger T, Reindl M (2017) Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases Front Immunol, 8: 529 | ||
| 2017 Book Chapters | |||
| o | Finsterer, J; Pakozdy, A; Bradl, M (2017) Epilepsy in Humans and Animals: From Patients to Disease Models. In: Jensen-Jarolim, E. (Eds.), Comparative Medicine: disorders linking humans with their animals Springer-Verlag : 13-25 | ||
| 2016 Original Articles | |||
| o | Zeka B, Hastermann M, Kaufmann N, Schanda K, Pende M, Misu T, Rommer P, Fujihara K, Nakashima I, Dahle C, Leutmezer F, Reindl M, Lassmann H, Bradl M (2016) Aquaporin 4-specific T cells and NMO-IgG cause primary retinal damage in experimental NMO/SD Acta Neuropathologica Communications, 4 (1): 82 | ||
| o | Oji S, Nicolussi E, Kaufmann N, Zeka B, Schanda K, Fujihara K, Illes Z, Dahle C, Reindl M, Lassmann H, Bradl M (2016) Experimental Neuromyelitis Optica Induces a Type I Interferon Signature in the Spinal Cord PLoS One, 11 (3): e0151244 | ||
| o | Schuh E, Berer K, Mulazzani M, Feil K, Meinl I, Lahm H, Krane M, Lange R, Pfannes K, Subklewe M, Gurkov R, Bradl M, Hohlfeld R, Kumpfel T, Meinl E, Krumbholz M (2016) Features of Human CD3+CD20+ T Cells J Immunol, 197 (4): 1111-1117 | ||
| 2016 Book Chapters | |||
| o | Bradl M, Lassmann H (2016) Neurologic autoimmunity mechanisms revealed by animal models. In: (Eds.), Handbook of Clinical Neurology : 121-143 | ||
| 2015 Original Articles | |||
| o | Licht-Mayer S, Wimmer I, Traffehn S, Metz I, Brück W, Bauer J, Bradl M, Lassmann H. (2015) Cell type-specific Nrf2 expression in multiple sclerosis lesions Acta Neuropathol, 130 (2): 263-277 | ||
| o | Zeka B, Hastermann M, Hochmeister S, Kögl N, Kaufmann N, Schanda K, Mader S, Misu T, Rommer P, Fujihara K, Illes Z, Leutmezer F, Sato D, Nakashima I, Reindl M, Lassmann H, Bradl M (2015) Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS Acta Neuropathol, 130 (6): 783-798 | ||
